Lennerz et al. found in his case series that three of five cases of MEC of cervix had rearrangement of the CRTC1 gene, but not MAML2, and 3 cases had amplification of MAML2, suggesting that other mechanisms may lead to tumorigenesis in those MECs without the canonical CRTC1/3::MAML2 fusions29. This evidence concerns the gene CRTC1 and mucoepidermoid carcinoma.