Other candidate tendinopathy genes associated with differing breed group risk of DSLD identified in this study include JUNB, and SEMA7A. The transcription factor JUNB has been linked to type I collagen disruption in fibrous connective tissue disease (Ponticos et al. 2015) and involvement in signaling pathways for tendon (Tan et al. 2020) and muscle (Verbrugge et al. 2018) homeostasis. Here, SEMA7A is linked to disease of the tendon.