Further, it promotes the release of proinflammatory factors such as chemokines, thymic stromal lymphopoietin (TSLP), IL-25, and granulocyte-macrophage colony-stimulating factor (GM-CSF), recruits inflammatory cells within the airway, and regulates the survival periods of different inflammatory cells, thus playing an important role in the asthma-related airway inflammatory response [26]. This evidence concerns the gene TSLP and asthma.