IFNG and myeloid sarcoma: It is important to note that CEF’s mechanism of action could be attributed to attenuation of inflammation-induced excitotoxicity associated with MS by slowing down the production of pro-inflammatory mediators (IL-17 and interferon gamma (IFNγ)) and proliferation of T cells (Melzer et al., 2008), as we discuss later in section “Attenuation of Pro-inflammatory Cytokines”.