There are currently many studies about the pathogenesis of CIDP; since the recognition of neurofascin-155 (NF-155), neurofascin-140 (NF-140), neurofascin-186 (NF-186), contactin-1 (CNTN-1), contactin-associated protein 1 (CASPR1), and other autoantibodies against the node of Ranvier or paranodal protein, we now have a better understanding of CIDP and promote the progress of diagnosis and treatment of CIDP (2, 3, 10, 12, 13). Here, CNTNAP1 is linked to chronic inflammatory demyelinating polyradiculoneuropathy.