Loss-of-function mutations in KCNQ2 and KCNQ3 channels occur in a small human population, causing a number of neurological disorders such as myokymia, neuromyotonia, neonatal-onset epilepsy, and epileptic encephalopathy [8, 14, 18, 20, 32]. The gene discussed is KCNQ2; the disease is Myokymia.