MCL1 and Miyoshi myopathy: In in vitro studies, bone marrow mesenchymal stem cell (BMSC)-derived exosomes could directly induce the survival of MM MDSCs and increase NO release from MDSCs by activating the STAT3 and STAT1 pathways and increasing the expression of anti-apoptotic proteins Bcl-xL and Mcl-1, thereby enhancing their suppressive function against T cells.