DNAJA3 and Parkinson disease: Recent studies show that upon assessment of sensory and motor function and brain region-specific expression levels by western blot analyses in control and PD rats, a 26 kDa breakdown product of the DnaJ fragment of hTid-1 was found in a 6-hydroxydopamine (6-OHDA) PD model of rats, in which gait symmetry, food handling, and sensory performance were found to be compromised [33].