Data pertaining to participant gender, age, BMI, smoking status, diabetes, hypertension, CRI, eGFR, D-dimer, FDP, CRP, abdominal vessel involvement, coronary malperfusion, mesenteric malperfusion, cerebral malperfusion, hypotension/shock, cardiac tamponade and operation status represented the stratification variables that were used to ascertain the corresponding development of effect sizes (Table 5). This evidence concerns the gene CRP and cardiac tamponade.