In a BALB/c mouse model of i.n. EV-D68 infection, simultaneous inhaled exposure to house dust mite—to induce an allergic airway response—resulted in increased recruitment of neutrophils and eosinophils to the airways and increased expression of proinflammatory and asthma-associated cytokines, interleukin-17A (IL-17A), CCL11, IL-5, and Muc5AC (34). This evidence concerns the gene IL17A and asthma.