Base on the western blot results, the TAMs which were cocultured with CCL2-overexpression GC cells showed a lower expression of iNOS, p-IKKβ, p-IκBα and p-p65, suggesting that CCL2 might decrease M1-like phenotype of TAMs via inhibiting NF-κB signaling (figure 2H). Here, NFKB1 is linked to gastric cancer.