The different characteristics of lipid metabolism during the latent (indicated by positive EBNA1-IgA) and lytic (indicated by high titers of EA-IgA and VCA-IgA) phases of EBV infection suggest that altered lipid metabolism in relation to or as a result of EBV reactivation might be an important mechanism linking together EBV infection, especially EBV reactivation, to the development of NPC. Here, CD79A is linked to Epstein-Barr virus infection.