Furthermore, individuals who continued to experience sequelae after infection (long COVID) had significantly higher levels of IL-17 and IL-2 and lower levels of Il-4 and IL-10, suggesting a possible “molecular signature” for long COVID characterized by a Th17 inflammatory profile with a reduced anti-inflammatory response mediated by IL-4 and IL-10, that must be confirmed by other studies enrolling a largest sample size. The gene discussed is IL17A; the disease is infection.