Current backbone treatment options include proteasome inhibitors (PIs; such as bortezomib, carfilzomib, and ixazomib), immunomodulatory agents (IMiDs; including thalidomide, lenalidomide, or pomalidomide), and anti‐CD38 monoclonal antibodies (mAbs; e.g., daratumumab), as well as histone deacetylase inhibitors (e.g., panobinostat), and most of which have been shown to extend survival in patients with MM [2, 3, 4, 5, 6, 7]. The gene discussed is CD38; the disease is Miyoshi myopathy.