Further gain- and loss-function experiments validated that overexpression of lncRNA FGD5-AS1 promoted the malignant phenotypes, including cell proliferation, invasion, migration, and tumorigenesis in CC cells in vitro and in vivo, while silencing of lncRNA FGD5-AS1 had opposite effects on the above cell functions, suggesting that lncRNA FGD5-AS1 acted as an oncogene to promote CC development, which was in accordance with the previous publications in other types of cancer [36]. The gene discussed is FGD5; the disease is cancer.