In addition, IR, a possible pathogenesis of NAFLD, had positive correlation with the raised plasma levels of apoB48-containing lipoproteins, which might be due to impaired hepatic ability of clearing TG-rich lipoproteins caused by excessive production of intestinal chylomicrons (CMs) and accumulation of serum CM remnants [13, 14]. Here, APOB is linked to metabolic dysfunction-associated steatotic liver disease.