To gain insight into why PRMT2-deficient myeloid cells fail to regress atherosclerosis compared to WT cells under nondiabetic conditions, we examined gene expression changes by performing bulk RNA sequencing (RNA-seq) on NG Ldlr−/−:WT and NG Ldlr−/−:Prmt2−/− plaque CD68+ cells collected by laser-capture microdissection20. This evidence concerns the gene PRMT2 and atherosclerosis.