AP1M2 and neoplasm: As a prelude to this analysis, we used the GEPIA webserver55 to perform a differential gene analysis of NIS (SLC5A5), μ1A (AP1M1), and μ1B (AP1M2) in samples of the Thyroid Cancer Genome Atlas.56 We found that SLC5A5 and AP1M1 are downregulated in tumor samples, and AP1M2 is upregulated with respect to control tissue (Supplementary Fig. S3).