IL17A and inflammatory bowel disease: The contribution of the IL-23/IL-17 axis has been recognized in the pathogenesis of IBD, as IL-17 induced by IL-23 elicits the release of proinflammatory cytokines, such as IL-6 and IL-8, releasing in myofibroblasts and epithelial cells which causes the recruitment of neutrophils and epithelial cell injury (45).