By FISH analysis (Table 1), NCOA2 rearrangement was positive in seven of the eight cases (7/8, 87.5%) (Figure 5A), and the mean percentage of NCOA2 split was 24% (range: 17% to 30%); the one negative for NCOA2 rearrangement (percentage of NCOA2 split: 2%) showed increased gene copy numbers of intact NCOA2 in 11% of the tumor cells (one to four additional fused signals per nucleus) (Figure 5B). This evidence concerns the gene NCOA2 and neoplasm.