However, it should be emphasized that NCOA2 rearrangement is not unique to SFAT, and NCOA2 can be the 3′ partner gene in fusions involved in many other bone and soft tissue neoplasms such as mesenchymal chondrosarcoma, alveolar rhabdomyosarcoma, congenital/infantile spindle cell rhabdomyosarcoma, biphenotypic sinonasal sarcoma, and uterine sarcoma with variable sex-cord differentiations (3, 4). This evidence concerns the gene NCOA2 and soft tissue neoplasm.