Some rearrangements preferentially occur at infancy, such as AML (megakaryoblastic) with t(1;22)(p13.3;q13.1); RBM15::MKL1, AML with t(7;12)(q36;p13); MNX1::ETV6, AML with t(8;16)(p11;p13); KAT6A::CREBBP (which can spontaneously remit in infancy, but has an intermediate prognosis in later childhood), and AML with inv(16)(p13.3q24.3); CBFA2R3::GLIS2 as reviewed by Quessada et al. [83]. Here, RBM15 is linked to acute myeloid leukemia.