Molecular mimicry of SARS-CoV-2 and acetylcholine receptor and cytokine storm due to TNF-α, IFN-γ, IL-6, regulatory T cell (Th-17), and IL-17 is contributed to the ARDS in COVID-19 and myasthenic crisis and also associated with the severity, poor outcome, and the mortality [11, 14, 39]. The gene discussed is IL17A; the disease is acute respiratory distress syndrome.