In addition to the therapeutic effect in AML cells, BCT-100 treatment can block the expression of cyclin D3 in cells, thereby inducing apoptosis of T- acute lymphoblastic leukemia (T-ALL) cells in vitro and in vivo, reflecting the potential therapeutic effect of BCT-100 on T-cell malignancies by consuming arginine (28). The gene discussed is CCND3; the disease is acute lymphoblastic leukemia.