As shown in Figure 6(f), the expression of p-AMPK and ULK protein was increased obviously, but the phosphorylation expression of mTOR protein was decreased, which further confirmed liensinine could induce autophagosome accumulation and suggested that liensinine might decrease the bioenergetic activity of NSCLC cells via impacting the AMPK/mTOR pathway. The gene discussed is MTOR; the disease is non-small cell lung carcinoma.