In addition, overexpressed LINC00336 acted as a crucial ferroptosis inhibitor in lung cancer by lowering cellular iron, lipid peroxidation, and mitochondrial superoxide through ELAV-like RNA-binding protein 1 (ELAVL1) interactivity, a novel regulator of ferroptosis (Wang et al., 2019). This evidence concerns the gene ELAVL1 and lung carcinoma.