We show that: 1) some of these genes remained upregulated 5 months after infection (LYZ, PLAC8 [fold increase 1.6; p=0.07)], 2) were upregulated during severe disease and downregulated 5 months after recovery (PFKB3, CXCL8 (fold decrease 1.93; p=0.06), TREML4, THBS1, EREG), or 3) remained downregulated long-term (IL1B, NLRP3) (77–81). This evidence concerns the gene THBS1 and infection.