TP53 and hepatitis B virus infection: KEGG pathway enrichment analysis further demonstrated that ‘viral carcinogenesis’, ‘Hepatitis B’, ‘cell cycle’, and ‘p53 signalling pathway’ terms were significantly enriched in the HBV‐infected liver tissues (Figure 1D), which was consistent with the result of our previous study, which showed that HBx could regulate cell cycle arrest and apoptosis through regulating p‐p53Thr55 dephosphorylation by B56γ.10