We recently described that FPR1 stimulation induces a proresolving program that relies on the expression of enzymes involved in SPM production (ALOX5, ALOX15A and ALOX15B), the release of specific SPMs (RvD1 and LXB4) and the expression of SPM receptors (GPR32, ChemR23 and BLT1) in gastric cancer (GC) cells [11]. This evidence concerns the gene FPR1 and gastric cancer.