APOB and familial hyperaldosteronism: Amongst patients for whom genetic testing results were available, only 21% had a mutation affecting the LDLR, apoB, or PCSK9 genes, which is lower than the rates of 69–77% reported previously in German patients with probable/definitive FH [27] and closer to the rates of 23–35% reported for patients with suspected/possible FH [27,28].