Finally, Kent et al. [11], in their meta-analysis, point out that metabolic pathways known to be dysregulated in GDM share a common downstream target of the TSHR, which activates cyclic adenosine monophosphate (cAMP), and, for its part, cAMP would also be involved in the altered endocrine function of syncytiotrophoblasts within the placenta. Here, TSHR is linked to gestational diabetes.