All this information, as well as our findings of positive correlations of OLFM2 in the liver with glucose and cholesterol levels and hepatic FAS, corroborates our previous hypothesis that hepatic expression of OLFM2 increases as NAFLD progresses, when at the same time, the metabolic imbalance also worsens [44]. This evidence concerns the gene FAS and metabolic dysfunction-associated steatotic liver disease.