NFKB1 and acute kidney injury: In a gentamicin-induced AKI model, paricalcitol restored impaired renal function by inhibiting renal inflammation and fibrosis via the interruption of the nuclear factor-kappaB (NF-κB)/extracellular signal-regulated kinase (ERK) signaling pathway and preservation of tubular epithelial integrity via the inhibition of the epithelial–mesenchymal transition (EMT) process [30].