Constitutive expression of the ALK gene via fusion with another gene within chromosome number 2 can promote the growth of tumors with abnormal ALK expression [9].The echinodermic microtubule-associated protein-like-4 (EML4)–ALK fusion gene was discovered in NSCLC patients in 2007 [10]. Here, EML4 is linked to non-small cell lung carcinoma.