CTSK and Miyoshi myopathy: The inhibition of TRPV2 channels by the antagonist SKF96365 in MM cells co-cultured with the monocyte/macrophage-like Raw264.7 cells reduces the expression of the osteoclast markers matrix metalloproteinase 9 (MMP9) and cathepsin K (CTSK), as a sign of decreased MM cells-induced osteoclastogenesis [61], arguing for the relevance of TRPV2 in bone disease.