The administration of exogenous oxytocin was found to attenuate normal bias in mentalizing and the processing of facial emotion, salience, aversion, uncertainty, and ambiguity in the social stimuli of persons with schizophrenia, particularly that which contributes to negative symptoms, through reduced activation in the brain networks of the amygdala, temporoparietal junction, posterior cingulate cortex, precuneus, and insula [222,223]. Here, OXT is linked to schizophrenia.