By mediating the stability of diaphanous-related formin 1 antisense RNA 1 (DIAPH1-AS1) in an m6A-dependent manner, WTAP promotes the formation of MTDH-LASP1 complex and the expression of LASP1, facilitating the cell growth of nasopharyngeal carcinoma [58]. This evidence concerns the gene LASP1 and nasopharyngeal carcinoma.