The differentially expressed genes identified here were related to relevant biological processes and participated in process networks and pathway maps associated with metabolic and neurodegenerative diseases, such as “TNF-alpha, IL-1 beta induces dyslipidemia and inflammation in obesity and type 2 diabetes in adipocytes” and “HSP70 and HSP40-dependent folding in Huntington’s disease”. This evidence concerns the gene IL1B and metabolic syndrome.