ALB and metabolic dysfunction-associated steatotic liver disease: An increase of Nfkbiz mRNA in the liver was not observed in Alb-Cre;Nfkbizfl/fl after CDAHFD challenge (Fig. 1C), which suggests that the induction of Nfkbiz in the liver during NAFLD development was mainly derived from albumin-producing hepatocytes; thus, other cells, including Kupffer cells, endothelial cells, and hepatic stellate cells, were not associated with the increase in IκBζ in this challenge.