The GO analysis indicated that PTCD1-related DEGs was highly abundant in some terms including “extracellular matrix organization,” “neutrophil degranulation,” “cell-matrix adhesion,” and “cell-substrate junction.” Genetic and epigenetic alterations might cause changed molecular pathways concerned tumor procession and, afterwards, force bladder cancer cells towards epithelial-to-mesenchymal transition (EMT) [27, 28]. The gene discussed is PTCD1; the disease is urinary bladder carcinoma.