AF G262 had the largest discrepancy among the antifibrotic groups in terms of the number of inflammatory cells (0.63) and the TGF-β1 (3.80) and NF-κB (0.48) levels, followed by AF G131, which also had the largest discrepancy among the antifibrotic groups in terms of NF-κB (0.48), TIMP-1 (11.74), and collagen I (14.50) levels. This evidence concerns the gene NFKB1 and atrial fibrillation.