Transposons likely play a role in the IPF pathogenesis,since familial IPF is most often caused by mutations in thegenes maintaining telomeres (the TERC and TERT genes)(Tsakiri et al., 2007; Fernandez et al., 2012), while sporadicforms of IPF are associated with polymorphic variants of thesegenes (Allen et al., 2020). Here, TERC is linked to idiopathic pulmonary fibrosis.