Aside from some known pathways supposedly involving gp96, such as the unfolded protein response, epithelial-mesenchymal transition, angiogenesis, and apoptosis [11, 13], the most intriguing finding was that inflammation-related pathways, such as the IFN-α response, the IL6-JAK-STAT6 signaling pathway, and the IFN-γ response, were highly enriched in gp96-related DEGs (Figures 6(a) and 6(b), Supplemental Figures 7(c) and 7(d)), suggesting an important role of gp96 in regulating the inflammatory response in glioma. This evidence concerns the gene STAT6 and glioma.