Patients suffering from Kawasaki disease (Ko et al., 2015), a condition that shows vasculitis of the coronary arteries and often results in coronary artery aneurysms or myocardial infarctions (Newburger et al., 2016), show an increased level of CXCL10 along with CXCR3-activated T cells in the plasma, suggesting CXCR3-CXCL10 as a potential therapeutic target to suppress progression of the cardiac diseases (Ko et al., 2015). This evidence concerns the gene CXCR3 and myocardial infarction.