CPT1A and acute myeloid leukemia: These data are consistent with similar findings in AML cells, which also showed a strong dependence on β-oxidation and in which inhibition of β-oxidation by etomoxir or ST1326, inhibitors of carnitine palmitoyltransferase 1a (CPT1A), resulted in an antiproliferative effect, induction of apoptosis and sensitized the cells to small molecule inhibitors [36–38].