NEUROG2 and glioblastoma: Apart from diverse chemotherapeutictargets, reprogramming of GBMcells has also emerged as a potential approach that promotes the differentiationof GBM cells to neuron-like cells through transcription factor-mediatedreprogramming.326 Notably, Asc1, Brn2,and Ngn2 (ABN) were found to be predominant transcription factorsthat abruptly reduced the growth of GBM cells in vitro and in vivo and promoted the conversion of GBMcells to non-divisible neurons.327 Recently,a study revealed the potential of small molecules to reprogram GBMcells.