However, the weight of evidence supports an interpretation that anosmia results predominantly from SARS-CoV-2 infection of non-neuronal cells in the olfactory epithelium and olfactory bulb (54), and dysgeusia more likely results from peripheral damage to ACE-2-expressing cells of taste buds and peripheral chemoreceptors, or cranial nerves responsible for gustation (CN VII, IX, or X) (55). The gene discussed is ACE2; the disease is Kallmann syndrome.