EIF4A2 and acute myeloid leukemia: Strikingly, knockdown of EIF4A2 significantly inhibited AML cell growth (Supplementary Fig. 5a, b), induced myeloid differentiation (Fig. 6f, Supplementary Fig. 5c–d) and resulted in increased cell apoptosis (Fig. 6g, Supplementary Fig. 5e–g) as compared to a shscramble control.