CSNK1D and myotonic dystrophy type 1: DM1 hiNeurons were successful in modelling aberrant splicing of MBNL1 exon 5 and MBNL2 exons 5 and 8, MAPT exon 2, CSNK1D exon 9 and MPRIP exon 9 as observed in post-mortem DM1 brain but to a lesser extent than what was previously reported for the last three mentioned transcripts [11, 22, 48, 50].