Table 1 shows that a higher prevalence of myocardial infarction was found (p = 0.015) in the vulnerable plaque group, and the TG, TC and LDL-C levels were significantly higher (p = 0.030, p < 0.001, and p < 0.001, respectively) than the non-vulnerable plaque group. The vulnerable plaque group had greater levels of tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) in the laboratory than the non-vulnerable plaque group (both of p = 0.001). This evidence concerns the gene TNF and myocardial infarction.