On the other hand, Thuringer et al. (2017) demonstrated an oncogene role for hsa-miR-5096 whose high expression contributed to increased invasiveness of glioblastoma cells by decreasing Kir4.1 protein levels, a K+ channel involved in the ionic homeostasis in the brain (Thuringer et al., 2017). The gene discussed is KCNJ10; the disease is glioblastoma.